Quisqualis indica, Combretum indicum. 使 君 子 Shǐ jūn zǐ   Rangoon creeper fruit, Chinese honeysuckle  
The name "quisqualis" comes from the Latin words for "who" and "what kind," which indicate that there was originally some question as to whether the plant was a vine or shrub.
PART USED: Dried ripe fruit
Nature: Slightly warm, neutral   FLAVOR: Sweet, pleasant
FUNCTIONS
GROUP: Antihelminthics
1. Expel Worms. Eliminates malnutrition and kills intestinal parasites.[1]
INDICATIONS
1. Intestinal parasite infestation. Marasmus in children.[1] Roundworms, malnutrition in children.[2] Abdominal distension and pain, poor food digestion.[1]
PREPARATIONS: Decoction  Dried ripe fruit  5-10 g.
Decoction- Dried ripe fruit  4-9 g.[1] Dried ripe fruit 9-12 g.[2]
  
References
Inner Path can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.  
Research

Quisqualis indica Improves Benign Prostatic Hyperplasia by Regulating Prostate Cell Proliferation and Apoptosis.
Ub Wijerathne C, Park HS, Jeong HY, Song JW, Moon OS, Seo YW, Won YS, Son HY, Lim JH, Yeon SH, Kwun HJ.
Abstract
Quisqualis indica (QI) has been used for treating disorders such as stomach pain, constipation, and digestion problem. This study was aimed to evaluate the therapeutic efficacy of QI extract on treating benign prostatic hyperplasia (BPH) in LNCaP human prostate cancer cell line and a testosterone-induced BPH rat model. LNCaP cells were treated with QI plus testosterone propionate (TP), and androgen receptor (AR) and prostate specific antigen (PSA) expression levels were assessed by Western blotting. To induce BPH, the rats were subjected to a daily subcutaneous injection of TP (3 mg/kg) for 4 weeks. The rats in treatment group were orally gavaged with QI (150 mg/kg) together with the TP injection. In-vitro studies showed that TP-induced increases in AR and PSA expression in LNCaP cells were reduced by QI treatment. In BPH-model rats, the prostate weight, testosterone in serum, dihydrotestosterone (DHT) concentration and 5α-reductase type 2 mRNA expression in prostate tissue were significantly reduced following the treatment with QI. TP-induced prostatic hyperplasia and the expression of proliferating cell nuclear antigen (PCNA) and cyclin D1 were significantly attenuated in QI-treated rats. In addition, QI induced apoptosis by up-regulating caspase-3 and -9 activity and decreasing the B-cell lymphoma 2 (Bcl-2)/Bcl-2-associated X protein (Bax) ratio in prostate tissues of BPH rats. Further investigation showed that TP-induced activation of AKT and glycogen synthase kinase 3β (GSK3β) was reduced by QI administration. Therefore, our findings suggest that QI attenuates the BPH state in rats through anti-proliferative and pro-apoptotic activities and might be useful in the clinical treatment of BPH.
PMID: 28943529 DOI: 10.1248/bpb.b17-00468 Biol Pharm Bull. 2017 Dec 1;40(12):2125-2133. doi: 10.1248/bpb.b17-00468. Epub 2017 Sep 22.

ncbi.nlm.nih.gov